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779423

Sigma-Aldrich

Ammonia solution

4 M in methanol

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Linear Formula:
NH3
CAS Number:
Molecular Weight:
17.03
MDL number:
PubChem Substance ID:
NACRES:
NA.21

form

liquid

Quality Level

concentration

3.6-4.4 M (back titration with NaOH)
3400-4400 mmol/L (back titration with NaOH)
4 M in methanol

SMILES string

N

InChI

1S/H3N/h1H3

InChI key

QGZKDVFQNNGYKY-UHFFFAOYSA-N

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1 of 4

This Item
341428294993392685
Ammonia solution 4 M in methanol

779423

Ammonia solution

Ammonia solution 2.0 M in methanol

341428

Ammonia solution

Ammonia anhydrous, ≥99.98%

294993

Ammonia

Ammonia solution 2.0 M in ethanol

392685

Ammonia solution

Quality Level

200

Quality Level

200

Quality Level

200

Quality Level

100

form

liquid

form

liquid

form

liquefied gas

form

liquid

General description

Ammonia solution (ammonia in methanol) is a colorless gas with a characteristic strong odor. It is highly soluble in water, alcohol, chloroform ether, ethanol, and methanol. Ammonia solution is commonly used as a base.

signalword

Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Chronic 3 - Eye Dam. 1 - Flam. Liq. 2 - Skin Corr. 1B - STOT SE 1

target_organs

Eyes

supp_hazards

Storage Class

3 - Flammable liquids

wgk_germany

WGK 2

flash_point_f

57.2 °F - open cup

flash_point_c

14 °C - open cup


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Eagleson M.
Concise Encyclopedia Chemistry, 65-66 (1994)
Ledgard J.
The Preparatory Manual of Explosives., 41-41 (2007)
Marion Vallet et al.
Kidney international, 88(1), 137-145 (2015-03-12)
Recent studies suggest that alkalinizing treatments improve the course of chronic kidney disease (CKD), even in patients without overt metabolic acidosis. Here, we tested whether a decreased ability in excreting urinary acid rather than overt metabolic acidosis may be deleterious
Olivier Braissant et al.
Journal of inherited metabolic disease, 36(4), 595-612 (2012-10-31)
Hyperammonemia can be caused by various acquired or inherited disorders such as urea cycle defects. The brain is much more susceptible to the deleterious effects of ammonium in childhood than in adulthood. Hyperammonemia provokes irreversible damage to the developing central
Lars Nybo et al.
The Journal of physiology, 563(Pt 1), 285-290 (2004-12-22)
We evaluated whether peripheral ammonia production during prolonged exercise enhances the uptake and subsequent accumulation of ammonia within the brain. Two studies determined the cerebral uptake of ammonia (arterial and jugular venous blood sampling combined with Kety-Schmidt-determined cerebral blood flow;

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