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Showing 1-5 of 5 results for "144-62-7" within Papers
Tobias J Grob et al.
Lung cancer (Amsterdam, Netherlands), 79(3), 221-227 (2012-12-15)
Next to EGFR mutation, EGFR gene copy number evaluated by fluorescence in situ hybridization (FISH) emerged as a potential predictive marker for sensitivity to EGFR tyrosine kinase inhibitors, although controversial data exist. As the diagnostic accuracy of predictive biomarkers can
Emilie Ceraudo et al.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 26(5), 2060-2071 (2012-02-01)
Vasoactive intestinal peptide (VIP) plays a major role in pathophysiology. Our previous studies demonstrated that the VIP sequence 6-28 interacts with the N-terminal ectodomain (N-ted) of its receptor, VPAC1. Probes for VIP and receptor antagonist PG97-269 were synthesized with a
Ralph Adams et al.
mAbs, 8(7), 1336-1346 (2016-06-18)
We generated an anti-albumin antibody, CA645, to link its Fv domain to an antigen-binding fragment (Fab), thereby extending the serum half-life of the Fab. CA645 was demonstrated to bind human, cynomolgus, and mouse serum albumin with similar affinity (1-7 nM), and
M A Khamashta et al.
The New England journal of medicine, 332(15), 993-997 (1995-04-13)
The antiphospholipid-antibody syndrome is a thrombophilic disorder in which venous or arterial thrombosis, or both, may occur in patients with antiphospholipid antibodies. The optimal treatment of these patients is unclear. We assessed the efficacy of warfarin, low-dose aspirin, or both
K N Pandey et al.
Endocrinology, 140(11), 5112-5119 (1999-10-28)
Mice lacking the gene (Npr1) encoding the natriuretic peptide receptor A (NPRA) have hypertension with elevated blood pressure and cardiac hypertrophy. In particular, Npr1 gene-deficient male mice exhibit lethal vascular events similar to those seen in untreated human hypertensive patients.
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