796832

Applied Filters:

Keyword:'796832'

Showing 1-9 of 9 results for "796832" within Papers

Reactive Extraction Strategy for Synthesizing Dioctyl Phthalate Intensified by Bifunctional Deep Eutectic Solvent [Im: 2PTSA]

Qin Hao, et al.

Chemical Engineering and Processing, 157, 108060-108060 (2020)

Exposure-based assessment of chemical teratogenicity using morphogenetic aggregates of human embryonic stem cells.

Yusuke Marikawa et al.

Reproductive toxicology (Elmsford, N.Y.), 91, 74-91 (2019-11-13)

Pluripotent stem cells recapitulate many aspects of embryogenesis in vitro. Here, we established a novel culture system to differentiate human embryonic stem cell aggregates (HESCA), and evaluated its utility for teratogenicity assessment. Culture of HESCA with modulators of developmental signals

Recovery From a Myocardial Infarction Is Impaired in Male C57bl/6 N Mice Acutely Exposed to the Bisphenols and Phthalates That Escape From Medical Devices Used in Cardiac Surgery.

Jijun Shang et al.

Toxicological sciences : an official journal of the Society of Toxicology, 168(1), 78-94 (2018-11-07)

Bisphenols and phthalates leach from medical devices, and this exposure is likely to increase in postcardiac surgery patients. Previous studies suggest that such chemical exposure may impact recovery and wound healing, yet the direct effects of bisphenols and phthalates are

Androgenic/antiandrogenic activities of PAEs determined by a novel AR-mediated reporter gene assay based on LLC-MK2 cells.

Xiaoju Ma et al.

Toxicology research, 8(4), 522-530 (2019-08-02)

Recent reports have concentrated on some androgens/antiandrogens and confirmed that certain chemicals have demonstrated androgenic/antiandrogenic activities in vitro. However, it is still unknown whether more chemicals in the human environment possess endocrine toxicity. 58A novel AR-mediated reporter gene assay based

The oxidative stress responses caused by phthalate acid esters increases mRNA abundance of base excision repair (BER) genes in vivo and in vitro.

Chunjiao Lu et al.

Ecotoxicology and environmental safety, 208, 111525-111525 (2020-10-30)

The base excision repair (BER) pathway is an important defense response to oxidative DNA damage. It is known that exposures to phthalate esters (PAEs), including Dibutyl phthalate (DBP), Mono-(2-ethylhexyl) phthalate (MEHP), and Di-(2-ethylhexyl) phthalate (DEHP), cause reactive oxygen species-induced DNA

The Effects of Mono-(2-Ethylhexyl) Phthalate (MEHP) on Human Estrogen Receptor (hER) and Androgen Receptor (hAR) by YES/YAS In Vitro Assay.

Da-Hye Kim et al.

Molecules (Basel, Switzerland), 24(8) (2019-04-24)

Endocrine active compounds with structural similarities to natural hormones such as 17β-estradiol (E2) and androgen are suspected to affect the human endocrine system by inducing hormone-dependent effects. This study aimed to detect the (anti-)estrogenic and (anti-)androgenic activities of mono-(2-ethylhexyl) phthalate

Mono-(2-ethylhexyl) phthalate induces oxidative stress and lipid accumulation in zebrafish liver cells.

Chang Gyun Park et al.

Comparative biochemistry and physiology. Toxicology & pharmacology : CBP, 230, 108704-108704 (2020-01-14)

The purpose of the present study was to examine the antioxidant and oxidative stress changes in zebrafish liver (ZFL) cells in the presence of mono-(2-ethylhexyl) phthalate (MEHP). When reactive oxygen species (ROS) and antioxidant levels were measured by immunoassay, significant

In Situ Fluorescence Tracking Toxic Metabolite Mono-2-ethylhexyl phthalate (MEHP) of Di-(2-ethylhexyl) phthalate (DEHP) in HeLa Cells

Huo Shao-Jie, et al.

Chemical Research in Toxicology, 32(10), 2006-2015 (2019)

Low Dose of Genistein Alleviates Mono-(2-Ethylhexyl) Phthalate-Induced Fetal Testis Disorder Based on Organ Culture Model.

Tong-Dian Zhang et al.

Oxidative medicine and cellular longevity, 2020, 4569268-4569268 (2020-06-23)

Mono-(2-ethylhexyl) phthalate (MEHP) and genistein have been classified as endocrine-disrupting chemicals (EDCs) which interfere with the differentiation and development of the male reproductive system. However, how these two EDCs would affect fetal rat testis development at a low dose was

Page 1 of 1