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Showing 1-11 of 11 results for "A6469" within Papers
Jiming Yin et al.
Cell death & disease, 9(10), 1031-1031 (2018-10-12)
The epithelial-mesenchymal transition (EMT) of podocytes had been reported to be involved in the glomerular fibrosis in diabetic kidney diseases, which was regulated by TGFβ and NFκB pathways. And apelin, an adipokine which is upregulated in diabetic kidney diseases, was
Bin Li et al.
The Journal of endocrinology, 249(1), 1-18 (2021-01-29)
Microcirculatory injuries had been reported to be involved in diabetic cardiomyopathy, which was mainly related to endothelial cell dysfunction. Apelin, an adipokine that is upregulated in diabetes mellitus, was reported to improve endothelial cell dysfunction and attenuate cardiac insufficiency induced
Apelin in disease
Antushevich H and Wojcik M
Clinica Chimica Acta; International Journal of Clinical Chemistry (2018)
Apelin protects against myocardial ischemic injury by inhibiting dynamin-related protein 1
Xu W, et al.
Oncotarget, 8(59), 100034-100034 (2017)
Apelin promotes diabetic nephropathy by inducing podocyte dysfunction via inhibiting proteasome activities.
Guo C
Journal of Cellular and Molecular Medicine, 19(9), 2273-2285 (2015)
Ewa Mlyczyńska et al.
International journal of molecular medicine, 45(3), 691-702 (2020-01-11)
Placentation requires the production of numerous growth factors, hormones and transcription factors. Many of them, like the adipose tissue‑derived leptin or adiponectin, have been identified in the placenta and their role has been established in the proliferation and subsequent development
D K Lee et al.
Journal of neurochemistry, 74(1), 34-41 (2000-01-05)
The apelin peptide was recently discovered and demonstrated to be the endogenous ligand for the G protein-coupled receptor, APJ. A search of the GenBank databases retrieved a rat expressed sequence tag partially encoding the preproapelin sequence. The GenBank search also
K Tatemoto et al.
Biochemical and biophysical research communications, 251(2), 471-476 (1998-10-30)
In the search for an endogenous ligand of the orphan G protein-coupled receptor APJ, the presence of the ligand in various tissue extracts was examined by measuring the increase in extracellular acidification rate of the cells expressing the APJ receptor
M X Zou et al.
FEBS letters, 473(1), 15-18 (2000-05-10)
The orphan G protein-coupled receptor APJ has been shown to be a coreceptor for human and simian immunodeficiency virus (HIV and SIV) strains. We have determined that some HIV and SIV strains use APJ as a coreceptor to infect the
Ewa Mlyczyńska et al.
International journal of molecular sciences, 22(5) (2021-04-04)
Previously, we demonstrated the expression of apelin and G-protein-coupled receptor APJ in human placenta cell lines as well as its direct action on placenta cell proliferation and endocrinology. The objective of this study was to examine the effect of apelin
B A Puffer et al.
Virology, 276(2), 435-444 (2000-10-21)
APJ is a seven transmembrane domain G-protein-coupled receptor that functions as a coreceptor for some primate immunodeficiency virus strains. The in vivo significance of APJ coreceptor function remains to be elucidated, however, due to the lack of an antibody that
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