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Showing 1-30 of 130 results for "C-016" within Papers
Christian Ginzkey et al.
Toxicology letters, 229(1), 303-310 (2014-07-02)
Human nasal miniorgan cultures (MOC) are a useful tool in ecogenotoxicology. Repetitive exposure to nicotine showed reversible DNA damage, and stable CYP2A6 expression was demonstrated in nasal MOC in previous investigations. The aim of the present study was to evaluate
Jinny E Sánchez-Rodríguez et al.
Environmental research, 136, 227-233 (2014-12-03)
Anti-smoking legislation has been associated with an improvement in health indicators. Since the cadmium (Cd) body burden in the general population is markedly increased by smoke exposure, we analyzed the impact of the more restrictive legislation that came into force
Kuan-Yen Tung et al.
Environmental research, 124, 7-12 (2013-04-30)
Environmental tobacco smoke (ETS) is a hazardous component of indoor air, and may increase the risk of respiratory diseases, atherosclerosis and otitis media in children. In this study, we explored the relationship between time inside the house, ETS exposure and
Ines Moreno-Gonzalez et al.
Nature communications, 4, 1495-1495 (2013-02-21)
Several epidemiological studies have shown that cigarette smoking might alter the incidence of Alzheimer's disease. However, inconsistent results have been reported regarding the risk of Alzheimer's disease among smokers. Previous studies in experimental animal models have reported that administration of
V Haufroid et al.
International archives of occupational and environmental health, 71(3), 162-168 (1998-05-20)
A minireview is presented concerning the use of cotinine as a tobacco-smoke exposure index. First, general considerations about methods for the determination of urinary cotinine are presented. Besides pure analytical aspects, this minireview considers major problems encountered in the establishment
Gonca Yilmaz et al.
Journal of tropical pediatrics, 59(5), 365-371 (2013-05-18)
Little research has focused on brief and practical strategies for addressing environmental tobacco smoke exposure through interventions focused explicitly on creating a smoke-free home. We used a two-group (intervention and control groups) repeated-measures randomized controlled trial design. Families were randomized
A P Santos-Silva et al.
The Journal of endocrinology, 218(1), 13-24 (2013-04-23)
Children from pregnant smokers show more susceptibility to develop obesity in adult life. Previously, we failed to demonstrate a program for obesity in rat offspring only when the mothers were exposed to tobacco smoke during lactation. Here, we studied the
Steven L Bramer et al.
Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals, 8(3-4), 187-203 (2003-08-29)
Subjects enrolled in studies are not always screened for routine habits such as smoking. Personal history is not always reliable and therefore an objective biomarker is necessary to screen for smokers. The objectives of this article were to review the
Johnny L Carson et al.
Inhalation toxicology, 25(3), 134-140 (2013-02-21)
Investigations of cell/molecular level effects of in vivo exposure of airway mucosa of experimental animals to common irritant gases have demonstrated structural and physiological changes reflective of breaches in epithelial barrier function, presence of inflammatory cell infiltrate and compromised ciliary
Observations on the number of saliva cotinine positives over a nine-year period.
S Mortlock et al.
British journal of biomedical science, 70(1), 43-44 (2013-04-27)
Van T Tong et al.
Acta obstetricia et gynecologica Scandinavica, 94(1), 106-111 (2014-10-29)
Evidence of bias of self-reported smoking cessation during pregnancy is reported in high-income countries but not elsewhere. We sought to evaluate self-reported smoking cessation during pregnancy using biochemical verification and to compare characteristics of women with and without biochemically confirmed
Maksymilian Kulza et al.
Przeglad lekarski, 69(10), 837-840 (2013-02-21)
The use of tobacco is a very serious threat to public health. Reducing the proportion of smokers easily leads to improved health of the general population. Smoking is a proven risk factor for respiratory disease, cardiovascular disease and cancer and
Erin K Zinkhan et al.
Pediatric research, 76(1), 17-23 (2014-04-15)
Maternal tobacco smoke (MTS) predisposes human and rat offspring to visceral obesity in early adulthood. Glucocorticoid excess also causes visceral obesity. We hypothesized that in utero MTS would increase visceral adiposity and alter the glucocorticoid pathway in young adult rats.
Cotinine as a biomarker of environmental tobacco smoke exposure.
N L Benowitz
Epidemiologic reviews, 18(2), 188-204 (1996-01-01)
Sungroul Kim et al.
Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco, 15(9), 1608-1616 (2013-03-20)
Cutoff values for distinguishing smokers from nonsmokers have generally been derived from studies in White populations. Even though recent population-based studies have demonstrated that cutoff values can differ by race and ethnicity, few studies have explored cutoff values among Asian
Natalie S Shenker et al.
Epidemiology (Cambridge, Mass.), 24(5), 712-716 (2013-07-23)
Most biomarkers of exposure tend to have short half-lives. This includes cotinine, a metabolite of nicotine widely used to assess smoke exposure. Cotinine is thus unsuitable as a determinant of past exposure to cigarette smoke. We used bisulphite pyrosequencing of
Sarah Connor Gorber et al.
Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco, 11(1), 12-24 (2009-02-28)
Smoking is a leading cause of premature mortality and preventable morbidity. Surveillance is most often based on self-reported data, but studies have shown that self-reports tend to underestimate smoking status. This study systematically reviewed the literature to measure the concordance
Marzenna Broszkiewicz et al.
Przeglad lekarski, 69(10), 846-853 (2013-02-21)
Diagnostics in the intensive interventions of tobacco use and dependence should include, among others assessments of nicotine dependence, motivation to quit and readiness to change. Based on the own research findings critical researcher's assessment of the accuracy of some recommended
Jakub Marcin Nowak et al.
Postepy higieny i medycyny doswiadczalnej (Online), 66, 996-1005 (2012-01-01)
This review presents the current state of knowledge on cotinine, the major metabolite of nicotine. Special attention is paid to the formation of this compound in the organism, its metabolism, application in diagnostic procedures and evaluation of its in vitro
Sunyoung Park et al.
Biochemical and biophysical research communications, 450(1), 13-18 (2014-05-27)
Although many peptides have therapeutic effects against diverse disease, their short half-lives in vivo hurdle their application as drug candidates. To extend the short elimination half-lives of therapeutic peptides, we developed a novel delivery platform for therapeutic peptides using an
R R Watts et al.
Environmental health perspectives, 84, 173-182 (1990-03-01)
A two-day technical workshop was convened November 10-11, 1986, to discuss analytical approaches for determining trace amounts of cotinine in human body fluids resulting from passive exposure to environmental tobacco smoke (ETS). The workshop, jointly sponsored by the U.S. Environmental
The detection of biomarkers of tobacco smoke exposure in wound fluid: a pilot study.
Amy Anne D Lassig et al.
Plastic and reconstructive surgery, 132(5), 892e-894e (2013-10-30)
T Berman et al.
Environment international, 59, 478-484 (2013-08-22)
The Ministry of Health Biomonitoring Study estimated exposure of individuals in the Israeli population to bisphenol A (BPA), organophosphate (OP) pesticides, phthalates, cotinine, polycyclic aromatic hydrocarbons (PAHs), and the phytoestrogenic compounds genistein and daidzein. In 2011, 250 individuals (ages 20-74)
Valentina Echeverria et al.
CNS neuroscience & therapeutics, 18(7), 517-523 (2012-04-26)
Tobacco smoking has been correlated with a lower incidence of Alzheimer's disease (AD). This negative correlation has been attributed to nicotine's properties. However, the undesired side-effects of nicotine and the absence of clear evidence of positive effects of this drug
Irene Shu et al.
Journal of chromatography. B, Analytical technologies in the biomedical and life sciences, 928, 139-145 (2013-05-02)
Concentrations of nicotine and its metabolites in blood are indicative of patients' current tobacco exposure, and their quantifications have been clinically applied to multiple assessments including demonstration of abstinence prior to heart-lung transplantation. For the purpose of transplant evaluation, the
[Cotinine].
Noriaki Takeda
Nihon rinsho. Japanese journal of clinical medicine, 62 Suppl 12, 502-508 (2005-01-22)
Water pipe smoking: not risk free.
Judy Peres
Journal of the National Cancer Institute, 106(8), doi:10-doi:10 (2014-08-15)
Elizabeth Hernández-Alvídrez et al.
The Journal of asthma : official journal of the Association for the Care of Asthma, 50(4), 347-353 (2013-02-13)
Passive smoking is associated with poor asthma control in children, but the mechanism is unknown. Leukotrienes are involved in the asthma pathogenesis and their synthesis is increased in adult subjects who actively smoke. To evaluate whether passive smoking, as assessed
Janani Sivasithamparam et al.
Cancer, 119(13), 2419-2426 (2013-04-12)
Use of tobacco products, excessive alcohol consumption, and high-risk sexual behaviors increase the risk of developing head and neck cancer and impacts treatment effectiveness after diagnosis. This study examined smoking and engagement in other modifiable behavioral risk factors and human
Carmen Iñiguez et al.
American journal of epidemiology, 178(7), 1067-1075 (2013-09-07)
In utero tobacco exposure has been associated with fetal growth restriction, but uncertainty remains about critical windows of exposure and specific effects on body segments. In the present study, we aimed to examine the association of maternal smoking with fetal
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