Search Within
MAB3228
Keyword:'MAB3228'
Showing 1-7 of 7 results for "MAB3228" within Papers
Reversible p53 inhibition prevents cisplatin ototoxicity without blocking chemotherapeutic efficacy.
EMBO molecular medicine, 9(1), 7-26 (2016-10-30)
Cisplatin is a widely used chemotherapy drug, despite its significant ototoxic side effects. To date, the mechanism of cisplatin-induced ototoxicity remains unclear, and hearing preservation during cisplatin-based chemotherapy in patients is lacking. We found activation of the ATM-Chk2-p53 pathway to
Identification of pulmonary Oct-4+ stem/progenitor cells and demonstration of their susceptibility to SARS coronavirus (SARS-CoV) infection in vitro.
Proceedings of the National Academy of Sciences of the USA null
American journal of physiology. Lung cellular and molecular physiology, 293(3), L740-L752 (2007-07-10)
It has been suggested that some adult bone marrow cells (BMC) can localize to the lung and develop tissue-specific characteristics including those of pulmonary epithelial cells. Here, we show that the combination of mild airway injury (naphthalene-induced) as a conditioning
International journal of molecular medicine, 37(6), 1521-1527 (2016-04-29)
The 293 cell line, used extensively in various types of studies due to the ease with which these cells can be transfected, was thought to be derived by the transformation of primary cultures of human embryonic kidney cells with sheared adenovirus
Homeodomain interacting protein kinase 2 regulates postnatal development of enteric dopaminergic neurons and glia via BMP signaling.
The Journal of Neuroscience null
Journal of neurosurgery. Spine, 21(3), 386-393 (2014-06-07)
Chordoma cells can generate solid-like tumors in xenograft models that express some molecular characteristics of the parent tumor, including positivity for brachyury and cytokeratins. However, there is a dearth of molecular markers that relate to chordoma tumor growth, as well
The Prostate, 75(14), 1620-1631 (2015-07-16)
The presence of inflammation in prostate cancer (PCa) and benign prostate hyperplasia (BPH) has been well described but the cellular mechanisms by which inflammation modulates the prostate are currently unclear. Prostate stem cells (PSC) not only maintain prostate homeostasis but
Page 1 of 1