SAB4200497

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Showing 1-7 of 7 results for "SAB4200497" within Papers

Thy-1, a versatile modulator of signaling affecting cellular adhesion, proliferation, survival, and cytokine/growth factor responses.

Tanya A Rege et al.

Biochimica et biophysica acta, 1763(10), 991-999 (2006-09-26)

Thy-1 is a 25-37 kDa glycosylphosphatidylinositol (GPI)-anchored protein involved in T cell activation, neurite outgrowth, apoptosis, tumor suppression, wound healing, and fibrosis. To mediate these diverse effects, Thy-1 participates in multiple signaling cascades. In this review, we discuss Thy-1 signaling

Thy-1 regulates fibroblast focal adhesions, cytoskeletal organization and migration through modulation of p190 RhoGAP and Rho GTPase activity.

Thomas H Barker et al.

Experimental cell research, 295(2), 488-496 (2004-04-20)

The precise biological role of Thy-1, a glycophosphatidyl-inositol (GPI)-linked cell surface glycoprotein in non-caveolar lipid raft microdomains, remains enigmatic. Evidence suggests that Thy-1 affects intracellular signaling through src-family protein kinases, and modulates adhesive and migratory events, such as thymocyte adhesion

Physical linkage of the fragile site FRA11B and a Jacobsen Syndrome chromosome deletion breakpoint in 11q23. 3

Jones C, et al.

Human Molecular Genetics, 3, 2123-2130 (1994)

Significance of CD90+ cancer stem cells in human liver cancer.

Zhen Fan Yang et al.

Cancer cell, 13(2), 153-166 (2008-02-05)

This study characterized cancer stem cells (CSCs) in hepatocellular carcinoma (HCC) cell lines, tumor specimens, and blood samples. The CD90+ cells, but not the CD90(-) cells, from HCC cell lines displayed tumorigenic capacity. All the tumor specimens and 91.6% of

THY1 is a candidate tumour suppressor gene with decreased expression in metastatic nasopharyngeal carcinoma.

Hong Lok Lung et al.

Oncogene, 24(43), 6525-6532 (2005-07-12)

Using oligonucleotide microarray analysis, THY1, mapping close to a previously defined 11q22-23 nasopharyngeal carcinoma (NPC) critical region was identified as showing consistent downregulated expression in the tumour segregants, as compared to their parental tumour-suppressing microcell hybrids (MCHs). Gene expression and

Targeting Extracellular miR-21-TLR7 Signaling Provides Long-Lasting Analgesia in Osteoarthritis.

Naoya Hoshikawa et al.

Molecular therapy. Nucleic acids, 19, 199-207 (2019-12-17)

Osteoarthritis (OA) is the most prevalent joint disorder associated with severe chronic pain. Although synovial inflammation is well correlated with pain severity, the molecular mechanism responsible for OA pain remains unclear. Here, we show that extracellular miR-21 released from synovial

A reduction in CD90 (THY-1) expression results in increased differentiation of mesenchymal stromal cells.

Moraes DA, et al.

Stem Cell Research & Therapy, 7 (2016)

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