SAB4500802

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Showing 1-8 of 8 results for "SAB4500802" within Papers

Akt1/PKBalpha is required for normal growth but dispensable for maintenance of glucose homeostasis in mice

Cho H, et al.

The Journal of Biological Chemistry (2001)

AKT plays a central role in tumorigenesis

Testa JR and Bellacosa A

Proceedings of the National Academy of Sciences of the USA (2001)

Autophagic and apoptotic cell death induced by the quinoline derivative 2-(6-methoxynaphthalen-2-yl)quinolin-4-amine in pancreatic cancer cells is via ER stress and inhibition of Akt/mTOR signaling pathway.

Habeebunnisa Begum et al.

Drug development research, 83(4), 910-926 (2022-01-30)

Pancreatic cancer (PC) is among the most lethal cancers and is resistant to existing therapies, which highlights the need for new and alternative therapeutic treatments. Autophagy is emerging as one of the alternative cell death mechanisms and is well known

ZP3 is Required for Germinal Vesicle Breakdown in Mouse Oocyte Meiosis

Gao LL, et al.

Scientific Reports (2017)

Association between single nucleotide polymorphisms in AKT1 and the risk of prostate cancer in the Chinese Han population

Liu JM, et al.

Genetics and molecular research : GMR (2017)

ZP3 is Required for Germinal Vesicle Breakdown in Mouse Oocyte Meiosis.

Lei-Lei Gao et al.

Scientific reports, 7, 41272-41272 (2017-02-02)

ZP3 is a principal component of the zona pellucida (ZP) of mammalian oocytes and is essential for normal fertility, and knockout of ZP3 causes complete infertility. ZP3 promotes fertilization by recognizing sperm binding and activating the acrosome reaction; however, additional

Sensitivity to chemotherapeutics of NSCLC cells with acquired resistance to EGFR-TKIs is mediated by T790M mutation or epithelial-mesenchymal transition.

Juan Zhou et al.

Oncology reports, 39(4), 1783-1792 (2018-02-03)

Chemotherapy is one of the methods to treat patients with non-small cell lung cancer (NSCLC) developing resistance to epidermal growth factor receptor (EGFR) tyrosine kinase inhibitors (TKIs), such as gefitinib. Previous studies revealed that the sensitivity to chemotherapy may depend

Dysregulation of microRNAs and renin-angiotensin system in high salt diet-induced cardiac dysfunction in uninephrectomized rats

Amara VR, et al.

PLoS ONE (2017)

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