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901903

Sigma-Aldrich

Sodium nitrite

anhydrous, Redi-Dri, ReagentPlus®, ≥99.0%

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Linear Formula:
NaNO2
CAS Number:
Molecular Weight:
69.00
MDL number:
NACRES:
NA.21

grade

anhydrous

vapor pressure

<0.0001 hPa ( 25 °C)

product line

ReagentPlus®
Redi-Dri

assay

≥99.0%

form

powder

autoignition temp.

914 °F

pH

9

mp

271 °C (lit.)

SMILES string

[Na+].[O-]N=O

InChI

1S/HNO2.Na/c2-1-3;/h(H,2,3);/q;+1/p-1

InChI key

LPXPTNMVRIOKMN-UHFFFAOYSA-M

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1 of 4

This Item
S2252799416237213
Sodium nitrite anhydrous, Redi-Dri&#8482;, ReagentPlus&#174;, &#8805;99.0%

901903

Sodium nitrite

-
Sodium nitrite ReagentPlus&#174;, &#8805;99.0%

S2252

Sodium nitrite

-
Sodium nitrite anhydrous, free-flowing, Redi-Dri&#8482;, ACS reagent, &#8805;97%

799416

Sodium nitrite

-
Sodium nitrite ACS reagent, &#8805;97.0%

237213

Sodium nitrite

Essential Grade
assay

≥99.0%

assay

≥99.0%

assay

≥97%

assay

≥97.0%

grade

anhydrous

grade

-

grade

ACS reagent, anhydrous

grade

ACS reagent

mp

271 °C (lit.)

mp

271 °C (lit.)

mp

271 °C (lit.)

mp

271 °C (lit.)

vapor pressure

<0.0001 hPa ( 25 °C)

vapor pressure

-

vapor pressure

<0.0001 hPa ( 25 °C)

vapor pressure

-

pH

9

pH

9 (20 °C, 100 g/L)

pH

9

pH

-

Legal Information

ReagentPlus is a registered trademark of Merck KGaA, Darmstadt, Germany
Redi-Dri is a trademark of Sigma-Aldrich Co. LLC

signalword

Danger

Hazard Classifications

Acute Tox. 3 Oral - Aquatic Acute 1 - Eye Irrit. 2 - Ox. Sol. 3

Storage Class

5.1B - Oxidizing hazardous materials

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable


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Paul J Shami et al.
Journal of medicinal chemistry, 49(14), 4356-4366 (2006-07-11)
The literature provides evidence that metabolic nitric oxide (NO) release mediates the cytotoxic activities (against human leukemia and prostate cancer xenografts in mice) of JS-K, a compound of structure R(2)N-N(O)=NO-Ar for which R(2)N is 4-(ethoxycarbonyl)piperazin-1-yl and Ar is 2,4-dinitrophenyl. Here
Rustem F Dautov et al.
Nitric oxide : biology and chemistry, 40, 36-44 (2014-05-27)
Previous studies in non-human blood vessels and in platelets have demonstrated that under hypoxic conditions release of NO from nitrite (NO2(-)) is potentiated by deoxyhaemoglobin. In the current study, we characterized hypoxic potentiation of NO2(-) effects in human vasculature and
Peter J Rix et al.
Clinical pharmacokinetics, 54(3), 261-272 (2014-11-26)
The efficacy of nebulized sodium nitrite (AIR001) has been demonstrated in animal models of pulmonary arterial hypertension (PAH), but it was not known if inhaled nitrite would be well tolerated in human subjects at exposure levels associated with efficacy in
Lucas C Pinheiro et al.
Free radical biology & medicine, 53(4), 701-709 (2012-06-23)
The new pathway nitrate-nitrite-nitric oxide (NO) has emerged as a physiological alternative to the classical enzymatic pathway for NO formation from l-arginine. Nitrate is converted to nitrite by commensal bacteria in the oral cavity and the nitrite formed is then
Borja Ibáñez et al.
Journal of the American College of Cardiology, 65(14), 1454-1471 (2015-04-11)
The damage inflicted on the myocardium during acute myocardial infarction is the result of 2 processes: ischemia and subsequent reperfusion (ischemia/reperfusion injury). During the last 3 decades, therapies to reduce ischemic injury (mainly reperfusion strategies) have been widely incorporated into

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