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120693

Sigma-Aldrich

α-Methyl-DL-tyrosine

98%

Synonym(s):

DL-2-Methyl-3-(4-hydroxyphenyl)alanine, AMPT

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About This Item

Linear Formula:
4-(HO)C6H4CH2C(CH3)(NH2)CO2H
CAS Number:
658-48-0
Molecular Weight:
195.22
Beilstein:
2938704
EC Number:
211-523-0
MDL number:
MFCD00004187
UNSPSC Code:
12352200
PubChem Substance ID:
24278151

Assay

98%

form

powder

reaction suitability

reaction type: solution phase peptide synthesis

technique(s)

HPLC: suitable

color

white to off-white

mp

>300 °C (lit.)

application(s)

peptide synthesis

SMILES string

CC(N)(Cc1ccc(O)cc1)C(O)=O

InChI

1S/C10H13NO3/c1-10(11,9(13)14)6-7-2-4-8(12)5-3-7/h2-5,12H,6,11H2,1H3,(H,13,14)

InChI key

NHTGHBARYWONDQ-UHFFFAOYSA-N

Gene Information

human ... TH(7054)

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Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Joram D Mul et al.
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The orexigenic neuropeptide melanin-concentrating hormone (MCH), a product of Pmch, is an important mediator of energy homeostasis. Pmch-deficient rodents are lean and smaller, characterized by lower food intake, body-, and fat mass. Pmch is expressed in hypothalamic neurons that ultimately
Khoa D Nguyen et al.
Nature, 480(7375), 104-108 (2011-11-22)
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The ability to respond to environmental temperature variation is essential for survival in animals. Flies show robust temperature-preference behaviour (TPB) to find optimal temperatures. Recently, we have shown that Drosophila mushroom body (MB) functions as a center controlling TPB. However
Maria Xilouri et al.
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The mechanisms through which aberrant alpha-synuclein (ASYN) leads to neuronal death in Parkinson's disease (PD) are uncertain. In isolated liver lysosomes, mutant ASYNs impair Chaperone Mediated Autophagy (CMA), a targeted lysosomal degradation pathway; however, whether this occurs in a cellular
Kinuyo Iwata et al.
The Journal of reproduction and development, 57(3), 379-384 (2011-03-02)
Ketosis is found in various pathophysiological conditions, including diabetes and starvation, that are accompanied by suppression of gonadal activity. The aim of the present study was to determine the role of ketone body in the brain in regulating pulsatile luteinizing
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