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Merck
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문서

CLS3422

Corning® Transwell® 24 well plates

8.0 μm pore size, Tissue Culture (TC)-treated surface round wells, clear bottom, clear wells, flat bottom wells, clear polystyrene (plate or dish), polycarbonate membrane, sterile, case of 48, pack of 12

동의어(들):

cell culture inserts, corning plates, tissue culture plate, tissue culture plates

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About This Item

UNSPSC 코드:
41122107
NACRES:
NB.13

product name

Corning® Transwell® polycarbonate membrane cell culture inserts, 6.5 mm Transwell with 8.0 μm pore polycarbonate membrane insert, TC-treated, w/lid, sterile, 48/cs

material

clear bottom
clear polystyrene (plate or dish)
clear wells
flat bottom wells
polycarbonate membrane
round wells

무균

sterile

특징

lid
skirt

포장

pack of 12
case of 48

제조업체/상표

Corning 3422

기술

cell culture | mammalian: suitable

삽입물 작업 용적

0.1 mL

멤브레인 직경

6.5 mm

크기

24 wells

표면적

0.33 cm2 , cell growth area

웰 작업 용적

0.6 mL

색상

translucent

공극 크기

8.0 μm

결합 유형

Tissue Culture (TC)-treated surface

유사한 제품을 찾으십니까? 방문 제품 비교 안내

일반 설명

  • 10 μm thick translucent polycarbonate membrane
  • Treated for optimal cell attachment
  • Packaged 12 inserts in a 24 well plate, 4 plates per case
  • Membrane must be stained for cell visibility
  • Sterilized by gamma radiation
Transwell cell culture inserts are convenient, easy-to-use permeable support devices for the study of both anchorage-dependent and anchorage-independent cell lines. They feature a 10 μm thin transparent polycarbonate membrane available in a variety of pore sizes. Transwell are treated for optimal cell attachment. Cells must be stained for cell visibility. The PC membrane is compatible with most organic fixatives and stains.

애플리케이션

Transwell was used during Boyden chamber assay to separate cells from upper and lower compartments.

법적 정보

Corning is a registered trademark of Corning, Inc.
Transwell is a registered trademark of Corning, Inc.

시험 성적서(COA)

제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.

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Driver genes with a mutually exclusive mutation pattern across tumor genomes are thought to have overlapping roles in tumorigenesis. In contrast, we show here that mutually exclusive prostate cancer driver alterations involving the ERG transcription factor and the ubiquitin ligase
James J Hoy et al.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 34(10), 13900-13917 (2020-08-25)
The PKA-inhibitor (PKI) family members PKIα, PKIβ, and PKIγ bind with high affinity to PKA and block its kinase activity, modulating the extent, and duration of PKA-mediated signaling events. While PKA is a well-known regulator of physiological and oncogenic events

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