GF27009514
Iron
rod, 150mm, diameter 5.0mm, as drawn, 99.99+%
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All Photos(2)
Synonym(s):
Iron, FE007943
Linear Formula:
Fe
CAS Number:
Molecular Weight:
55.85
MDL number:
UNSPSC Code:
12141721
PubChem Substance ID:
NACRES:
NA.23
assay
99.99%
form
rod
manufacturer/tradename
Goodfellow 270-095-14
resistivity
9.71 μΩ-cm
L × diam.
150 mm × 5.0 mm
bp
2750 °C (lit.)
mp
1535 °C (lit.)
density
7.86 g/mL at 25 °C (lit.)
SMILES string
[Fe]
InChI
1S/Fe
InChI key
XEEYBQQBJWHFJM-UHFFFAOYSA-N
Related Categories
General description
For updated SDS information please visit www.goodfellow.com.
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50 years ago in the Journal of Pediatrics: Iron metabolism in premature infants: II. Prevention of iron deficiency.
Pamela J Kling
The Journal of pediatrics, 164(4), 768-768 (2014-03-22)
Thomas A Russo et al.
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our
Laura M van Staalduinen et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), 5171-5176 (2014-04-08)
The enzymes PhnY and PhnZ comprise an oxidative catabolic pathway that enables marine bacteria to use 2-aminoethylphosphonic acid as a source of inorganic phosphate. PhnZ is notable for catalyzing the oxidative cleavage of a carbon-phosphorus bond using Fe(II) and dioxygen
Antonino Missineo et al.
Infection and immunity, 82(6), 2448-2459 (2014-04-02)
Staphylococcus lugdunensis is a coagulase-negative staphylococcus that is a commensal of humans and an opportunistic pathogen. It can cause a spectrum of infections, including those that are associated with the ability to form biofilm, such as occurs with endocarditis or
Florence Lima et al.
Environmental and molecular mutagenesis, 55(3), 266-277 (2014-04-12)
DNA methylation is an epigenetic mechanism that drives phenotype and that can be altered by environmental exposures including radiation. The majority of human radiation exposures occur in a relatively low dose range; however, the biological response to low dose radiation
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