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Key Documents

HAWP29325

Millipore

MF-Millipore Membrane

pore size 0.45 μm, filter diam. 293 mm, hydrophilic

Synonym(s):

MF-Millipore Membrane, mixed cellulose esters, Hydrophilic, 0.45 μm, 293 mm, white, plain

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About This Item

UNSPSC Code:
40161507
eCl@ss:
32031602

material

mixed cellulose esters (MCE) membrane
plain filter
white filter

Quality Level

100

feature

hydrophilic

manufacturer/tradename

MF-Millipore™

parameter

4 L/min-cm2 air flow rate
60 mL/min-cm2 water flow rate
75 °C max. temp.

filter diam.

293 mm

thickness

180 μm

gravimetric extractables

2.5%

refractive index

n/D 1.51

matrix

MF-Millipore™

pore size

0.45 μm pore size
79 % porosity

capacity

262 μg/cm2 adsorption capacity (insulin)

bubble point

≥2.2 bar, air with water at 23 °C

shipped in

ambient

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General description

Filter Code: HAWP
Filter Type: Screen filter

Other Notes

Directions for Use

  • Organism Retention: Microorganism
  • Mode of Action: Filtration (size exclusion)
  • Application: BioProcessing
  • Intended Use: Reduction or removal of microorganism/bioburden
  • Instructions for Use: Please refer first page of Visual Inspection Guide
  • Storage Statement: Store in dry location
  • Disposal Statement: Dispose of in accordance with applicable federal, state and local regulations.

Legal Information

MF-Millipore is a trademark of Merck KGaA, Darmstadt, Germany

pictograms

Flame
GHS02

signalword

Danger

hcodes

H228

Hazard Classifications

Flam. Sol. 1

Storage Class

4.1B - Flammable solid hazardous materials

wgk_germany

WGK 3

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Diogo B Peruchetti et al.
Archives of biochemistry and biophysics, 674, 108115-108115 (2019-09-29)
Evidence points to a possible role of tubular sodium reabsorption in worsening renal injury. Proximal tubule (PT) albumin overload is a critical process in the development of tubule-interstitial injury (TII), and consequently in progression of renal disease. We studied the

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