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18-1630

Sigma-Aldrich

Lithium standard solution

suitable for atomic absorption spectrometry, 1000 ppm Li, 1 mg/mL Li

Synonym(s):

Lithium chloride solution

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About This Item

Linear Formula:
LiCl
CAS Number:
7447-41-8
Molecular Weight:
42.39
MDL number:
MFCD00011078
UNSPSC Code:
12352302
PubChem Substance ID:
329751641

form

liquid

availability

available only in Japan

concentration

1 mg/mL Li
1000 ppm Li

suitability

suitable for atomic absorption spectrometry

storage temp.

15-25°C

SMILES string

[Li+].[Cl-]

InChI

1S/ClH.Li/h1H;/q;+1/p-1

Inchi Key

KWGKDLIKAYFUFQ-UHFFFAOYSA-M

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pictograms

Corrosion
GHS05

signalword

Warning

hcodes

H290

Hazard Classifications

Met. Corr. 1

Storage Class

8B - Non-combustible corrosive hazardous materials

wgk_germany

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificates of Analysis (COA)

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Otilia Zarnescu et al.
Micron (Oxford, England : 1993), 46, 66-75 (2013-01-29)
In the present study we examined the effects of lithium chloride on the Corydoras aeneus caudal fin regeneration. After caudal fin amputation, the fish were exposed 3h daily to 35 mM lithium chloride for 9 days. The effects of lithium
Li-Wei Zheng et al.
International journal of oral science, 5(1), 1-6 (2013-03-30)
This study aimed to compare epithelial cells derived from human embryonic stem cells (hESCs) to human ameloblast-lineage cells (ALCs), as a way to determine their potential use as a cell source for ameloblast regeneration. Induced by various concentrations of bone
Aitor González et al.
PloS one, 8(1), e53323-e53323 (2013-01-18)
The mouse segmentation is established from somites, which are iteratively induced every two hours from the presomitic mesoderm (PSM) by a system known as the segmentation clock. A crucial component of the segmentation clock is the gene Hes7, which is
Weiwei Gao et al.
Carcinogenesis, 34(6), 1208-1215 (2013-02-08)
We recently reported that an N-terminally truncated retinoid X receptor-α (tRXRα) produced in cancer cells acts to promote cancer cell growth and survival through AKT activation. However, how RXRα is cleaved and how the cleavage is regulated in cancer cells
A Briolay et al.
Biochemical and biophysical research communications, 430(3), 1072-1077 (2012-12-26)
Although anti-tumor necrosis factor (TNF)-α treatments efficiently block inflammation in ankylosing spondylitis (AS), they are inefficient to prevent excessive bone formation. In AS, ossification seems more prone to develop in sites where inflammation has resolved following anti-TNF therapy, suggesting that
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