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- Air Pollution Drives Macrophage Senescence through a Phagolysosome-15-Lipoxygenase Pathway: This study reveals that fine dust, particularly PM2.5, can induce cellular aging in macrophages via specific biochemical pathways, suggesting implications for chronic respiratory diseases and highlighting the importance of air quality control (Thomas et al., 2024).
- Perceived occupational health risks, noise and dust exposure levels among street sweepers in Mwanza City in Northern Tanzania: This research assesses the exposure of workers to fine dust and its perceived health impacts, underscoring the necessity for improved protective measures in occupational settings (Nyanza et al., 2024).
- Role of low-density lipoprotein in mediating the effect of air pollution on coronary heart disease: The study explores how fine dust influences heart disease through lipid pathways, contributing to a deeper understanding of environmental risk factors for cardiovascular conditions (Sun et al., 2024).
- Possible modulating functions of probiotic Lactiplantibacillus plantarum in particulate matter-associated pulmonary inflammation: Investigates the therapeutic potential of probiotics against inflammation caused by fine dust exposure, proposing a novel approach to mitigating air pollution′s health effects (Gupta et al., 2023).
- Source apportionment of fine atmospheric particles in Bloemfontein, South Africa, using positive matrix factorization: This study focuses on identifying the sources of fine dust, providing critical data for environmental policy and public health strategies (van der Westhuizen et al., 2024).
アナリシスノート
For more information please click on the below product number:
ERMCZ110
ERMCZ110
法的情報
ERM is a registered trademark of European Commission
保管分類コード
13 - Non Combustible Solids
WGK
WGK 3
引火点(°F)
Not applicable
引火点(℃)
Not applicable
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