ECM642
In Vitro Vascular Permeability Assay (96-well)
This In Vitro Vascular Permeability Assay kit employs a 96-well plate, and provides an efficient system for evaluating the effects of chemicals & drug compounds on endothelial cell adsorption, transport & permeability.
동의어(들):
Endothelial cell model for permeability assay
로그인조직 및 계약 가격 보기
모든 사진(2)
About This Item
UNSPSC 코드:
12352207
eCl@ss:
32161000
NACRES:
NA.32
추천 제품
Quality Level
종 반응성(상동성에 의해 예측)
all
제조업체/상표
Chemicon®
기술
cell based assay: suitable
검출 방법
fluorometric
배송 상태
wet ice
애플리케이션
Research Category
Cell Structure
Cell Structure
The Millipore® In Vitro Vascular Permeability Assay kit provides an efficient system for evaluating the effects of chemicals and drug compounds on endothelial cell adsorption, transport, and permeability. It is ideal for measuring compounds that may disrupt or protect an endothelial monolayer. Each In Vitro Vascular Permeability Assay Kit contains sufficient reagents for the evaluation of 96 samples. The In Vitro Vascular Permeability Assay Kit is intended for research use only, not for diagnostic or therapeutic applications.
성분
96-Well Permeability Insert Assembly, Collagen-coated: One 96-well receiver tray containing 96 connected porous cell culture inserts pre-coated with type I rat-tail collagen.
Receiver Tray, 96-Well: Two 96-well plates.
FITC-Dextran Solution: Two vials containing 250 µL.
Cell Stain: One bottle containing 10 mL.
Black Plate, 96-Well Opaque: One 96-well plate.
Receiver Tray, 96-Well: Two 96-well plates.
FITC-Dextran Solution: Two vials containing 250 µL.
Cell Stain: One bottle containing 10 mL.
Black Plate, 96-Well Opaque: One 96-well plate.
저장 및 안정성
Store kit materials at 2-8°C; use within 4 months from date of receipt. Do not freeze.
법적 정보
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Millipore is a registered trademark of Merck KGaA, Darmstadt, Germany
면책조항
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
Storage Class Code
10-13 - German Storage Class 10 to 13
WGK
WGK 2
시험 성적서(COA)
제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.
이미 열람한 고객
You-Hong Cheng et al.
PloS one, 12(11), e0187949-e0187949 (2017-11-11)
Recent evidence suggests that chemokine CXCL12, the cognate agonist of chemokine receptors CXCR4 and ACKR3, reduces thrombin-mediated impairment of endothelial barrier function. A detailed characterization of the effects of CXCL12 on thrombin-mediated human lung endothelial hyperpermeability is lacking and structure-function
Mohammad Shoeb et al.
Environmental research, 180, 108900-108900 (2019-11-13)
Inhalation of welding fume (WF) can result in the deposition of toxic metals, such as manganese (Mn), in the brain and may cause neurological changes in exposed workers. Alterations in telomere length are indicative of cellular aging and, possibly, neurodegeneration.
Kibbeum Song et al.
European journal of pharmacology, 869, 172891-172891 (2019-12-27)
The blood-brain barrier (BBB) is a highly selective permeability barrier that separates the circulating blood from the brain and extracellular fluid in the central nervous system (CNS). The BBB is formed by cerebral endothelial cells connected by tight junctions. Prion
Peng Sun et al.
The Journal of pharmacology and experimental therapeutics, 382(3), 266-276 (2022-07-03)
Diabetic nephropathy is a leading cause of end-stage renal disease, characterized by endothelial dysfunction and a compromised glomerular permeability barrier. Dysregulation of the angiopoietin 1 (ANGPT1)/angiopoietin 2 (ANGPT2) signaling axis is implicated in disease progression. We recently described the discovery
Guo-Xiang Ruan et al.
The EMBO journal, 31(7), 1692-1703 (2012-02-14)
Herein, we report that vascular endothelial growth factor A (VEGF-A) engages the PI3K/Akt pathway by a previously unknown mechanism that involves three tyrosine kinases. Upon VEGF-A-dependent activation of VEGF receptor-2 (VEGFR-2), and subsequent TSAd-mediated activation of Src family kinases (SFKs)
문서
Cell based angiogenesis assays to analyze new blood vessel formation for applications of cancer research, tissue regeneration and vascular biology.
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