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Merck
모든 사진(1)

주요 문서

ET0300

Sigma-Aldrich

E-TOXATE Kit

sufficient for 100 assays

동의어(들):

Endotoxin detection kit

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About This Item

UNSPSC 코드:
12352202
NACRES:
NA.84

사용

sufficient for 100 assays

Quality Level

저장 온도

−20°C

애플리케이션

E-TOXATE Kit has been used in the semiquantitative determination of endotoxin levels in 0.1 % (w/v) dispersions of polyelectrolyte nanoparticle construct (NPs) and 0.1 % (w/v) polymer solutions.

법적 정보

E-Toxate is a trademark of Sigma-Aldrich Co. LLC

키트 구성품 역시 별도로 이용 가능함

제품 번호
설명
SDS

  • E8029E-Toxate Endotoxin standard, for endotoxin quantitation 1 mL/vialSDS

  • E8779E-Toxate reagent from Limulus polyphemus, sufficient for 20 tests 5 vial(s)SDS

  • 2107E-Toxate Water, endotoxin, free 5 x 30SDS

관련 제품

호환 제품

제품 번호
설명
가격

Storage Class Code

10 - Combustible liquids


시험 성적서(COA)

제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.

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Intra-articular delivery of a nanocomplex comprising salmon calcitonin, hyaluronic acid, and chitosan using an equine model of joint inflammation.
Sladek S, et al.
Drug delivery and translational research, 8(5), 1421-1435 (2018)
Svenja Sladek et al.
Drug delivery and translational research, 8(5), 1421-1435 (2018-06-28)
Polyelectrolyte nanoparticle constructs (NPs) comprising salmon calcitonin (sCT), chitosan (CS), and hyaluronic acid (HA) were previously established as having anti-inflammatory potential when injected via the intra-articular (i.a.) route to a mouse model. We attempted to translate the formulation to a
Xue Q Liu et al.
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The development and maintenance of memory B cells (MBC) is dependent on germinal centres (GC) with follicular dendritic cell (FDC) networks. We have previously shown that FDC networks within GC of the spleen express a novel ligand for CD38 and
Atsuko Hayashida et al.
The American journal of pathology, 174(2), 509-518 (2009-01-17)
In pneumonia caused by the bacterium Staphylococcus aureus, the intense inflammatory response that is triggered by this infection can lead to the development of lung injury. Little is known, however, about the impact of specific virulence factors on this inflammatory

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