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Merck
모든 사진(1)

문서

N8641

Sigma-Aldrich

Nutrient Mixture F-12 Ham

HEPES modification, with sodium bicarbonate, without ʟ-glutamine, liquid, sterile-filtered, suitable for cell culture

동의어(들):

Ham’s F-12

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About This Item

MDL number:
UNSPSC 코드:
12352207
NACRES:
NA.75

product name

Nutrient Mixture F-12 Ham, HEPES Modification, with 25 mM HEPES and sodium bicarbonate, without L-glutamine, liquid, sterile-filtered, suitable for cell culture

Quality Level

무균

sterile-filtered

형태

liquid

기술

cell culture | mammalian: suitable

불순물

endotoxin, tested

pH

>7.2

성분

L-glutamine: no
sodium pyruvate: 0.11 g/L
NaHCO3: yes
phenol red: yes
HEPES: yes

배송 상태

ambient

저장 온도

2-8°C

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재구성

Supplement with 0.146 g/L L-glutamine.

Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 1

Flash Point (°F)

Not applicable

Flash Point (°C)

Not applicable


시험 성적서(COA)

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문서 라이브러리 방문

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Journal of proteome research, 18(9), 3521-3531 (2019-07-30)
Proteins are involved in all tasks of life, and their characterization is essential to understand the underlying mechanisms of biological processes. We present a method called "differential visual proteomics" geared to study proteome-wide structural changes of proteins and protein-complexes between
Tsuyoshi Mishiro et al.
The EMBO journal, 28(9), 1234-1245 (2009-03-27)
Long-range regulatory elements and higher-order chromatin structure coordinate the expression of multiple genes in cluster, and CTCF/cohesin-mediated chromatin insulator may be a key in this regulation. The human apolipoprotein (APO) A1/C3/A4/A5 gene region, whose alterations increase the risk of dyslipidemia
Fumitaka Kawakami et al.
PloS one, 7(1), e30834-e30834 (2012-02-04)
Leucine-rich repeat kinase 2 (LRRK2), a large protein kinase containing multi-functional domains, has been identified as the causal molecule for autosomal-dominant Parkinson's disease (PD). In the present study, we demonstrated for the first time that (i) LRRK2 interacts with tau
Henri J Huttunen et al.
Journal of molecular neuroscience : MN, 37(1), 6-15 (2008-07-12)
A common pathogenic event that occurs in all forms of Alzheimer's disease is the progressive accumulation of amyloid beta-peptide (Abeta) in brain regions responsible for higher cognitive functions. Inhibition of acyl-coenzyme A: cholesterol acyltransferase (ACAT), which generates intracellular cholesteryl esters

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