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Merck
모든 사진(1)

주요 문서

SAB1305552

Sigma-Aldrich

Anti-MAP1LC3A antibody

mouse monoclonal, 166AT1234

동의어(들):

Autophagy-related protein LC3 A, Autophagy-related ubiquitin-like modifier LC3 A, MAP1 light chain 3-like protein 1, MAP1LC3A, Microtubule-associated proteins 1A/1B light chain 3A

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모든 사진(1)

About This Item

UNSPSC 코드:
12352203
NACRES:
NA.41

제품명

MONOCLONAL ANTI-LC3 (APG8) antibody produced in mouse, clone 166AT1234, IgG fraction of antiserum, buffered aqueous solution

생물학적 소스

mouse

Quality Level

100

항체 형태

IgG fraction of antiserum

항체 생산 유형

primary antibodies

클론

166AT1234, monoclonal

양식

buffered aqueous solution

분자량

14272 Da

종 반응성

rat, mouse, human

기술

immunofluorescence: 1:25
immunohistochemistry: 1:50-1:100
western blot: 1:1000

동형

IgG1κ

NCBI 수납 번호

NP_115903.1

UniProt 수납 번호

Q9H492

배송 상태

wet ice

저장 온도

−20°C

타겟 번역 후 변형

unmodified

유전자 정보

human ... MAP1LC3A(84557)

물리적 형태

Supplied in PBS with 0.09% (W/V) sodium azide

면책조항

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class Code

10 - Combustible liquids

Flash Point (°F)

Not applicable

Flash Point (°C)

Not applicable

가장 최신 버전 중 하나를 선택하세요:

시험 성적서(COA)

Lot/Batch Number
SG190329AA
SD100625
SD110414A
SDG090127
SG100621AB

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특정 버전이 필요한 경우 로트 번호나 배치 번호로 특정 인증서를 찾을 수 있습니다.

COA 검색

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문서 라이브러리 방문

Matteo Bordoni et al.
Cells, 11(8) (2022-04-24)
Mitochondria alterations are present in tissues derived from patients and animal models, but no data are available for peripheral blood mononuclear cells (PBMCs) of ALS patients. This work aims to investigate mitophagy in PBMCs of sporadic (sALS) patients and how
Hong Ding et al.
Journal of asthma and allergy, 17, 717-731 (2024-08-06)
Accumulating evidence indicates that oxidative stress and inflammation are the pathological basis of allergic diseases. Inhibition of NOD-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome could ameliorate allergic rhinitis (AR). Here, we explored the effects and mechanisms that underlie NLRP3
Xiaoming Shu et al.
Molecular medicine reports, 16(2), 1180-1188 (2017-06-07)
Peripheral blood T lymphocytopenia has previously been identified in polymyositis/dermatomyositis (PM/DM) patients. Therefore, the present study aimed to examine the potential role of autophagy in peripheral blood T cell survival in PM/DM patients. Transmission electron microscopy was used to detect
Maomao Sun et al.
Frontiers in immunology, 12, 685523-685523 (2021-08-03)
Recent studies have shown that autophagy upregulation can attenuate sepsis-induced acute kidney injury (SAKI). The tumor suppressor p53 has emerged as an autophagy regulator in various forms of acute kidney injury (AKI). Our previous studies showed that p53 acetylation exacerbated
Rongrong Hua et al.
International journal of molecular sciences, 19(5) (2018-05-08)
We have reported that conventional protein kinase Cγ (cPKCγ)-modulated neuron-specific autophagy improved the neurological outcome of mice following ischemic stroke through the Akt-mechanistic target of rapamycin (mTOR) pathway. However, its detailed molecular mechanism remains unclear. In this study, primary cortical
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