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Merck
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주요 문서

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H4884

Sigma-Aldrich

Hippuryl-His-Leu acetate salt

동의어(들):

N-Benzoyl-Gly-His-Leu

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모든 사진(4)

About This Item

실험식(Hill 표기법):
C21H27N5O5
CAS Number:
103404-54-2
Molecular Weight:
429.47
UNSPSC 코드:
12352200
PubChem Substance ID:
329815212
NACRES:
NA.32

분석

≥98% (HPLC)

Quality Level

200

형태

powder

solubility

acetic acid: 50 mg/mL, clear, colorless to very faintly yellow

저장 온도

−20°C

SMILES string

CC(O)=O.CC(C)C[C@H](NC(=O)[C@H](Cc1c[nH]cn1)NC(=O)CNC(=O)c2ccccc2)C(O)=O

InChI

1S/C21H27N5O5.C2H4O2/c1-13(2)8-17(21(30)31)26-20(29)16(9-15-10-22-12-24-15)25-18(27)11-23-19(28)14-6-4-3-5-7-14;1-2(3)4/h3-7,10,12-13,16-17H,8-9,11H2,1-2H3,(H,22,24)(H,23,28)(H,25,27)(H,26,29)(H,30,31);1H3,(H,3,4)/t16-,17-;/m0./s1

InChI key

NSEVQQKNHZCIKL-QJHJCNPRSA-N

유전자 정보

human ... ACE(1636) , ACE2(59272)
mouse ... ACE3(217246)
rat ... ACE(11421) , ACE2(70008) , ACE3(498012)

Amino Acid Sequence

NBz-Gly-His-Leu

생화학적/생리학적 작용

Substrate for angiotensin converting enzyme.

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point (°F)

Not applicable

Flash Point (°C)

Not applicable

개인 보호 장비

Eyeshields, Gloves, type N95 (US)

시험 성적서(COA)

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Mammalian Genome null
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FEBS letters, 555(3), 597-600 (2003-12-17)
It was determined that flavan-3-ols and procyanidins have an inhibitory effect on angiotensin I converting enzyme (ACE) activity, and the effect was dependent on the number of epicatechin units forming the procyanidin. The inhibition by flavan-3-ols and procyanidins was competitive
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Mammalian genome : official journal of the International Mammalian Genome Society, 20(3), 170-179 (2009-03-04)
In humans, the insertion/deletion polymorphism in the angiotensin converting enzyme (ACE) gene accounts for half of the variance in plasma ACE activity. The deletion allele is associated with high plasma ACE activity, cardiovascular disease, and renal disease. In rat, a
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Journal of food science and technology, 57(4), 1371-1381 (2020-03-18)
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Human genetic data have associated angiotensin-converting enzyme (ACE) with Alzheimer disease (AD), and purified ACE has been reported to cleave synthetic amyloid beta-protein (Abeta) in vitro. Whether deficiency in ACE activity, arising from genetic alteration or pharmacological inhibition, can decrease
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